Tumor necrosis factor-α and CD80 modulate CD28 expression through a similar mechanism of T-cell receptor-independent inhibition of transcription

Keyphrases

• Tumor Necrosis Factor-α 100%
• T Cell Receptor 100%
• Receptor-independent 100%
• Transcription Inhibition 100%
• CD28 100%
• CD80 100%
• T Cell Lines 11%
• Replicative Senescence 11%
• Jtag 11%
• T Cells 5%
• Aging 5%
• Biochemical Processes 5%
• Ligation 5%
• Clonal Expansion 5%
• Reconstitution 5%
• Phorbol 12-myristate 13-acetate 5%
• Costimulatory Molecules 5%
• T Cell Immunity 5%
• Biological Outcomes 5%
• Transactivation 5%
• Ontogenesis 5%
• Chronic Inflammatory Diseases 5%
• Phenotypic Transition 5%
• Transient Expression 5%
• Null Cell 5%
• Cell Line Models 5%
• Repeated Stimulations 5%
• CD28null T Cells 5%
• Senescent T Cells 5%
• Chronic Infectious Diseases 5%

Immunology and Microbiology

• Tumor Necrosis Factor 100%
• T Cell Receptor 100%
• CD28 100%
• CD80 100%
• T Cell 35%
• Cell Line 17%
• Precursor 5%
• Adoptive Immunity 5%
• Dynamics 5%
• Ontogeny 5%
• Transactivation 5%
• Transient Expression 5%
• Null Cell 5%
• In Vitro 5%
• Acetate 5%