Mitochondrial dysfunction as a pathogenic mediator of chronic obstructive pulmonary disease and idiopathic pulmonary fibrosis

Keyphrases

• Mitochondria 100%
• Chronic Obstructive Pulmonary Disease 100%
• Idiopathic Pulmonary Fibrosis 100%
• Mitochondrial Dysfunction 100%
• Mitophagy 100%
• Mitochondrium 100%
• Chronic Lung Disease 75%
• Type-specific 50%
• Autophagy 50%
• Mouse Model 25%
• Disease Progression 25%
• Response to Injury 25%
• Inflammatory Response 25%
• Human Disease 25%
• Disease Pathogenesis 25%
• Cell Organelles 25%
• Cellular Response 25%
• Fibrosis 25%
• Intracellular Signaling 25%
• Subtype-specific 25%
• Genetic Studies 25%
• Cell Death Pathways 25%
• Human Studies 25%
• Emphysema 25%
• Fibrotic Response 25%
• Protein Turnover 25%
• Calcium Homeostasis 25%
• Necrotic Cells 25%
• Organelle Turnover 25%
• Adaptive Mechanism 25%
• Energy Generation 25%
• Necroptosis 25%
• Cellular Metabolism 25%
• Cellular Protection 25%
• Damage-associated Molecular Patterns 25%
• Selective Autophagy 25%
• Cell Death Program 25%
• Phosphatase and Tensin Homolog-induced Putative Kinase 1 25%

Medicine and Dentistry

• Mitochondrion 100%
• Mediator 100%
• Chronic Obstructive Pulmonary Disease 100%
• Idiopathic Pulmonary Fibrosis 100%
• Mitochondrial Disorder 100%
• Mitophagy 100%
• Chronic Lung Disease 75%
• Diseases 50%
• Autophagy 50%
• Cell Death 50%
• Phosphotransferase 25%
• Cell Metabolism 25%
• Emphysema 25%
• Fibrosis 25%
• Upregulation 25%
• Human Study 25%
• Intracellular Signaling 25%
• Calcium Homeostasis 25%
• Phosphatase 25%
• Tensin 25%
• Selective Autophagy 25%
• Necroptosis 25%

Pharmacology, Toxicology and Pharmaceutical Science

• Fibrosing Alveolitis 100%
• Chronic Obstructive Lung Disease 100%
• Chronic Lung Disease 75%
• Diseases 50%
• Phosphotransferase 25%
• Mouse Model 25%
• Fibrosis 25%
• Emphysema 25%
• Human Study 25%
• Phosphatase 25%
• Tensin 25%