Abstract

There is no cure for chronic rejection of transplanted organs. Macrophages are heavily involved in chronic rejection. Macrophages’ movement into the graft depends on the actin cytoskeleton and its regulators the GTPase RhoA and its effector ROCK kinase. In this chapter, we describe how the interference with macrophage RhoA/ROCK pathway disrupts macrophage actin cytoskeleton, impairing their functions and migration to the allograft, and preventing the development of chronic rejection, and how such interference can be applied to clinical therapies.

Original languageEnglish (US)
Title of host publicationResults and Problems in Cell Differentiation
PublisherSpringer Science and Business Media Deutschland GmbH
Pages135-150
Number of pages16
DOIs
StatePublished - 2026

Publication series

NameResults and Problems in Cell Differentiation
Volume77
ISSN (Print)0080-1844
ISSN (Electronic)1861-0412

Keywords

  • Actin
  • Allograft
  • Chronic rejection
  • Macrophage
  • Mechanotransduction
  • RhoA
  • ROCK

ASJC Scopus subject areas

  • Developmental Biology
  • Cell Biology

Divisions

  • Abdominal Transplant

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