Fractalkine signaling regulates the inflammatory response in an α-synuclein model of Parkinson disease

Keyphrases

• Inflammatory Response 100%
• Parkinson's Disease Model 100%
• Alpha-synuclein (α-syn) 100%
• Fractalkine 100%
• CX3CR1 100%
• Inflammation 80%
• Microglia 60%
• Phagocytosis 60%
• Neuroinflammation 40%
• Parkinson's Disease 40%
• Adeno-associated Viral Vector 40%
• Serotype 2 40%
• Neurodegeneration 20%
• Multiple Sclerosis 20%
• In Vivo Model 20%
• Neurodegenerative Diseases 20%
• Dopaminergic Neurons 20%
• Overexpression 20%
• MHC Class II 20%
• Microglial Cells 20%
• Dopamine Neurons 20%
• Substantia Nigra Pars Compacta 20%
• Alzheimer's Disease Model 20%
• Neurotoxin 20%
• Putative Drug Targets 20%
• Fluorescent Microspheres 20%
• Protein Internalization 20%
• Fluorescent Beads 20%
• IgG Deposition 20%
• IgG Expression 20%
• Chemokine CX3CL1 20%

Medicine and Dentistry

• Parkinson's Disease 100%
• Serositis 100%
• Synuclein 100%
• Fractalkine 100%
• Microglia 60%
• Phagocytosis 60%
• Serotype 40%
• Neuroinflammation 40%
• Adeno Associated Virus Vector 40%
• Neurodegeneration 20%
• In Vitro 20%
• Receptor 20%
• Microsphere 20%
• Mediator 20%
• Alzheimer's Disease 20%
• Neurodegenerative Disorder 20%
• Multiple Sclerosis 20%
• Substantia Nigra Pars Compacta 20%
• Dopaminergic Neuron 20%
• Neurotoxin 20%
• Internalization 20%
• Alpha Synuclein 20%

Neuroscience

• Parkinson's Disease 100%
• Synuclein 100%
• CX3CR1 100%
• Fractalkine 100%
• Microglia 60%
• Phagocytosis 60%
• Adeno Associated Virus Vector 40%
• Neurodegenerative Disorder 20%
• In Vivo 20%
• In Vitro 20%
• Receptor 20%
• Dopaminergic Neuron 20%
• Microsphere 20%
• Alzheimer's Disease 20%
• Neurodegeneration 20%
• Multiple Sclerosis 20%
• Substantia Nigra Pars Compacta 20%
• Internalization 20%
• Neurotoxin 20%
• Alpha-Synuclein 20%