Animal models of allergic bronchopulmonary aspergillosis

G. Grunig; D. B. Corry; R. L. Coffman; D. M. Rennick; V. P. Kurup

doi:10.1016/S0889-8561(05)70028-X

Animal models of allergic bronchopulmonary aspergillosis

G. Grunig, D. B. Corry, R. L. Coffman, D. M. Rennick, V. P. Kurup

Research output: Contribution to journal › Article › peer-review

9 Scopus citations

Abstract

Animal models of ABPA provide an intriguing insight into disease pathogenesis across various species. In the mouse, the availability of gene-targeted animals allows for a detailed dissection of critical cells and cytokines that mediate immunopathology. T cells are the central effectors that ultimately induce lung pathology and airway hyperreactivity in murine ABPA.

Original language	English (US)
Pages (from-to)	661-679
Number of pages	19
Journal	Immunology and Allergy Clinics of North America
Volume	18
Issue number	3
DOIs	https://doi.org/10.1016/S0889-8561(05)70028-X
State	Published - 1998

ASJC Scopus subject areas

Immunology and Allergy
Immunology

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10.1016/S0889-8561(05)70028-X

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title = "Animal models of allergic bronchopulmonary aspergillosis",

abstract = "Animal models of ABPA provide an intriguing insight into disease pathogenesis across various species. In the mouse, the availability of gene-targeted animals allows for a detailed dissection of critical cells and cytokines that mediate immunopathology. T cells are the central effectors that ultimately induce lung pathology and airway hyperreactivity in murine ABPA.",

author = "G. Grunig and Corry, {D. B.} and Coffman, {R. L.} and Rennick, {D. M.} and Kurup, {V. P.}",

note = "Funding Information: DNAX Research Institute of Molecular and Cellular Biology is supported by Schering–Plough Corporation. The work was partially supported by a US Veterans Affairs Medical Research Grant (VPK) and by Grant HL03344 (DBC) and Grant AI42349 (VPK) from the National Institutes of Health. Copyright: Copyright 2021 Elsevier B.V., All rights reserved.",

year = "1998",

doi = "10.1016/S0889-8561(05)70028-X",

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AU - Corry, D. B.

AU - Coffman, R. L.

AU - Rennick, D. M.

AU - Kurup, V. P.

N1 - Funding Information: DNAX Research Institute of Molecular and Cellular Biology is supported by Schering–Plough Corporation. The work was partially supported by a US Veterans Affairs Medical Research Grant (VPK) and by Grant HL03344 (DBC) and Grant AI42349 (VPK) from the National Institutes of Health. Copyright: Copyright 2021 Elsevier B.V., All rights reserved.

PY - 1998

Y1 - 1998

N2 - Animal models of ABPA provide an intriguing insight into disease pathogenesis across various species. In the mouse, the availability of gene-targeted animals allows for a detailed dissection of critical cells and cytokines that mediate immunopathology. T cells are the central effectors that ultimately induce lung pathology and airway hyperreactivity in murine ABPA.

AB - Animal models of ABPA provide an intriguing insight into disease pathogenesis across various species. In the mouse, the availability of gene-targeted animals allows for a detailed dissection of critical cells and cytokines that mediate immunopathology. T cells are the central effectors that ultimately induce lung pathology and airway hyperreactivity in murine ABPA.

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Animal models of allergic bronchopulmonary aspergillosis

Abstract

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