A mutant Ahr allele protects the embryonic kidney from hydrocarbon-induced deficits in fetal programming

Keyphrases

• C57BL 100%
• Hydrocarbons 100%
• Benzo(a)pyrene 100%
• Fetal Programming 100%
• Embryonic Kidney 100%
• Polymorphism 66%
• Developmental Programming 66%
• Nephrogenesis 66%
• Molecular Mechanism 33%
• Kidney Disease 33%
• Integration Site 33%
• Pathogenic Mechanism 33%
• Epidermal Growth Factor Receptor 33%
• Splice Variant 33%
• Growth Factors 33%
• Insulin-like 33%
• Renal Function 33%
• Adult-onset 33%
• Ligand Binding 33%
• E-cadherin 33%
• Developmental Disease 33%
• Pregnant Rats 33%
• Mutant Alleles 33%
• A1 Receptor 33%
• Fetal Origins 33%
• Transcriptional Targets 33%
• PAX2 33%
• Functional Links 33%
• Gestational Day 33%
• Wilms' Tumor Gene 33%
• Syndecan-1 33%
• Frizzled 33%
• Retinoic Acid Receptor 33%
• RNA Splice Sites 33%
• Experimental Model Systems 33%
• Metanephric 33%
• Homeobox Protein 33%
• Renal Development 33%
• Wnt4 33%
• Paired Box Gene 2 33%
• Dysmorphogenesis 33%
• Renal Morphogenesis 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Benzo(a)pyrene 100%
• C57BL 6 Mouse 66%
• Synapsin I 66%
• Diseases 33%
• Epidermal Growth Factor Receptor 33%
• Messenger RNA 33%
• Tumor Suppressor Protein 33%
• Kidney Disease 33%
• Mouse Mammary Tumor Virus 33%
• Uvomorulin 33%
• Nephroblastoma 33%
• Somatomedin C Receptor 33%
• Teratogenesis 33%
• Retinoic Acid Receptor Alpha 33%
• Syndecan 1 33%
• Homeodomain Protein 33%